PHD2 deletion in endothelial or arterial smooth muscle cells reveals vascular cell type-specific responses in pulmonary hypertension and fibrosis

Approved

Classifications

MinEdu publication type
A1 Journal article (peer-reviewed)
Definition
Article
Target group
Scientific
Peer reviewed
Peer-reviewed
Article type
Journal article
Host publication type
Journal

Authors of the publication

Number of authors
10
Authors
Elamaa, Harri; Kaakinen, Mika; Nätynki, Marjut; Szabo, Zoltan; Ronkainen, Veli-Pekka; Äijälä, Ville; Mäki, Joni M.; Kerkelä, Risto; Myllyharju, Johanna; Eklund, Lauri

Publication channel information

Title of journal/series
Angiogenesis
ISSN (print)
0969-6970
ISSN (electronic)
1573-7209
ISSN (linking)
0969-6970
Publisher
Kluwer academic publishers
Publication forum ID
51108
Publication forum level
1
Country of publication
Netherlands
Internationality
Yes

Detailed publication information

Publication year
2022
Reporting year
2022
Journal/series volume number
25
Journal/series issue number
2
Page numbers
259-274
DOI
10.1007/s10456-021-09828-z
Language of publication
English

Co-publication information

International co-publication
No
Co-publication with a company
No

Availability

Classification and additional information

MinEdu field of science classification
1182 Biochemistry, cell and molecular biology
Keywords
Pulmonary hypertension; Fibrosis; Basement membrane; Endothelial cell; Smooth muscle cell; Extracellular matrix; Hypoxia

Funding information

Funding information in the publication
Open Access funding provided by University of Oulu including Oulu University Hospital. Research was supported by the Academy of Finland Centre of Excellence Program (251314) for LE and JM, and the Academy Research Fellow Grant (136880) for LE.

Infrastructure information

Infrastructure information in the publication
Biocenter Oulu Transgenic-, Electron Microscopy-, and Light Microscopy Core Facilities and the Oulu Laboratory Animal Center supported by Biocenter Finland and the University of Oulu are acknowledged for their specific scientific expertise and research infrastructure services.

Source database ID

WoS ID
WOS:000740198600001
Scopus ID
2-s2.0-85122659438
Other database ID
PMID: 34997404